Pancuronium bromide CAS# 15500-66-0

17beta-dioldiacetatedimetho, Promotion Season Now in Store and Free Sample for Testing with Factory Price

• Chemical Name:  Pancuronium bromide
• CAS No.:     15500-66-0
• Molecular Fomula: ： C35H60Br2N2O4
• Molecular weight：732.67
• Appearance: colorless solution
• Sample：Available

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Description of 17beta-dioldiacetatedimetho

Pancuronium (trademarked as Pavulon) is a muscle relaxant with various medical uses. It is used in euthanasia and is the second of three drugs administered during most lethal injections in the United States.

Basic Info of 17beta-dioldiacetatedimetho

Basic Info
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Basic Info

Numbering system

Properties

Safety Info

What is Pancuronium bromide?

• With the progress of science and technology and the development of society, chemical products have invariably permeated our daily lives, in medicine, food, cosmetics, electronics, industry, and other areas, becoming an essential part of our lives. One such product is Bismuth Pancuronium bromide   which has developed particularly rapidly in recent years. Do you know about Bismuth Pancuronium bromide  ?
• The official answer：Pancuronium bromide, [5] chemically known as dibromo(1, 1′-[3α,17β-diacetoxy-5α-androstane-2β,16β-diyl]bis[1-methylpiperidine]), is a long-acting non-depolarizing myorelaxant in synthetic bisquaternium steroids and is the first myorelaxant used in ICU, its effect is stronger than that of Dextro Its effect is 4-5 times stronger than that of dextran, and it has a blocking effect on all M-choline receptors in the body. It is mainly used as an adjunct to general anesthesia, for tracheal intubation during general anesthesia and muscle relaxation during surgery. This product has the characteristics of no histamine release, no ganglion block, and does not cause hypotension.

What’s the application of Pancuronium bromide?

• When it binds to N2 choline receptors on skeletal muscle cells, it does not produce a depolarizing effect, but competitively blocks the depolarizing effect of the neurotransmitter acetylcholine, thus producing a relaxing effect on skeletal muscle. Its muscle relaxation efficacy is 3-5 times that of cartridge poison, and the effect appears more quickly. This product cannot be taken orally, but is only used for intravenous injection. The effect is visible in 3-4 minutes after injection, and the maintenance time is about 20-30 minutes. About 30% of the product is metabolized and inactivated in the liver, and most of it is excreted in its original form through the kidney. It is characterized by a moderate excitatory effect on the cardiovascular system compared with cartridge poisoning, while there is no significant histamine release, no accumulation with continuous use, and no adverse effects on the fetus. Neostigmine counteracts the inotropic effect of this product.

Conclusion

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